The orthomolecular understanding of postpartum psychosis (PPD) is that it may be caused by extreme excess retention of copper. During pregnancy, a woman’s copper levels will increase. In some cases, they can double. After childbirth, the copper levels in a typical woman will normalize naturally. Copper levels that do not normalize on their own in women who develop postpartum depression can ultimately develop postpartum psychosis. Excess copper in the brain can alter the balance of dopamine and norepinephrine, two chemicals that regulate our moods.
Females who are copper toxic have poor copper detox mechanisms. For these women, copper levels rise with each pregnancy, and they do not come down. These copper-toxic women with poor copper detox mechanisms find themselves depressed after pregnancy and unable to recover, even years later. Some of these females move from postpartum depression to postpartum psychosis. Women with postpartum depression after their first two children are at greater risk with subsequent pregnancies unless they receive proper treatment for their biochemical imbalance. It is not solely the number of pregnancies but the actual copper level that affects the risk for postpartum psychosis. In terms of postpartum psychosis (PPP), this approach focuses on providing nutrients to correct imbalances caused by hormone disruptions during and after pregnancy.